Interkinesis is a small period of rest that some cells may go through between meiosis I and meiosis II. Some cells may go through a brief period between these two parts of meiosis called interkinesis. This means that after meiosis I the two daughter cells do not re-enter interphase and no duplication event occurs between meiosis I and meiosis II.

How do we define meiosis II?Дirectly after meiosis I, the two haploid daughter cells with extra chromosome copies undergo meiosis II, so that the sister chromatids, or identical chromosome copies, can be split evenly to produce four haploid daughter cells (Fig. Following directly after meiosis I, meiosis II produces four haploid daughter cells, known as gametes. Meiosis II is the s econd round of cell division in the process of meiosis, or the creation of gametes (sex cells). If you haven't had the chance, go check out our article on m eiosis I before delving into this next big topic! If you started this article by first reading about meiosis I, then you are expecting the next step in the journey of meiosis. Image Source: Khan Academy.The best things in life come in pairs: best friends, milk and cookies, and meiosis I and meiosis II.

Condensation occurs parallel to the nuclear envelope assembly mediated by the Mitotic-Exit Network (MEN) Cyclin-Dependent Kinase dephosphorylation which restarts the interphase.ДЇigure: Telophase in mitosis.

The compact chromosomes are necessary for the cellвЂ™s preparation and initiation of the interphase.

Chromosome condensation (decompaction) forms expanded chromatin.

This influences the formation of the nuclear envelope and the double-layered nuclear membrane.

The nuclear membrane is absorbed by the endoplasmic membrane during metaphase however targeting the nuclear membrane protein-containing vesicles of the endoplasmic reticulum to the chromatins takes place during telophase.

These organelles are dismantled during prophase and metaphase phases.

This is the reconstruction of the double nuclear membrane, the formation of the nuclear pore complexes, and the formation of the internal nuclear lamina into the inner nuclear membrane.

This is the shortening of the kinetochore microtubules, pulling the chromosomes to the poles of the cell and separating from the chromosomes.

Cdc48 also triggers telophase mechanisms of spindle disassembly, nuclear envelope assembly, and chromosome condensation by activation ubiquitination in the proteasomes.

Distancing of chromosomes from the metaphase plate triggering early telophase.

It also mediated dephosphorylation by activating downstream regulatory proteins specific for telophase such as CHD1 dephosphorylation proteins which target proteolysis leading to the cellular switching activity, eventually leading to the G1 phase of interphase.

The restriction in the nucleus is later observed when it plays a major role in the Mitotic Exit Network (MEN) whereby it triggers spindle disassembly and the assembling of the nuclear envelope, during telophase.

Its initial function is during the early stages of anaphase where it plays a key role in stabilizing the mitotic spindles and releasing more Cdc-14 into the nucleus where it becomes restricted.

One of the most significant Cdks is the Cdc-14 which is initially activated by being released into the nucleus from the nucleolus and exported to the cytoplasm.

This allows for disassembling the spindles, mending the nuclear envelope, and chromosomal condensation of new daughter cells.

While dephosphorylation occurs as the mitotic cell cycle comes to an end in telophase.

Phosphorylation of the mitotic Cyclin-dependent Kinase substrate leads to the spindle assembly, chromosome condensation, and nuclear envelope breakdown which takes place n the initial stages of mitosis.

These include: Dephosphorylation of the mitotic Cyclin-Dependent Kinase substrate (Cdk) There are various mechanisms that take place within this phase which leads to the formation of new daughter cells. Telophase in mitosis is significantly an extension of anaphase and metaphase that is controlled by the dephosphorylation of the mitotic cyclin-dependent kinase (CDK) substrate.

Prophase ii